EVIDENCE BASE / PHASE 3

The trials that built the semaglutide evidence base

Nine pivotal programs, more than 25,000 patients, and a sequence of indications that has moved well beyond glycemic control. Here is what each trial actually measured — and what it found.

TRIALS / PIVOTAL 9+
PATIENTS >25K
FOLLOW-UP (LONGEST) 208wk
JOURNALS NEJM · JAMA · Lancet
Hexagonal grid of abstract clinical-trial data glyphs

Most recent: ESSENCE — semaglutide in MASH (2025)

The ESSENCE Phase 3 trial, published in the New England Journal of Medicine on June 5, 2025, was the first successful Phase 3 trial of a GLP-1 receptor agonist in metabolic dysfunction-associated steatohepatitis (MASH) — the inflammatory liver disease formerly called NASH[11].

TRIAL / PHASE 3 / 2025

ESSENCE Part 1

POPULATION
800 adults with biopsy-confirmed MASH and fibrosis stage 2 or 3
DOSE
subcutaneous 2.4 mg once weekly
DURATION
72 weeks (interim primary analysis)
PUBLICATION
NEJM · June 5, 2025
PRIMARY OUTCOMES

Resolution of steatohepatitis without worsening of fibrosis: 62.9% semaglutide vs 34.3% placebo. Fibrosis improvement without worsening of steatohepatitis: 36.8% vs 22.4%[11].

The regulatory submission was accepted with Priority Review. MASH is a leading driver of progressive liver disease in adults with obesity and type 2 diabetes, and prior to ESSENCE no GLP-1 RA had produced confirmatory Phase 3 histology data.

FLOW — kidney and cardiovascular outcomes in T2DM-CKD (2024)

The FLOW trial established semaglutide as the first GLP-1 receptor agonist with confirmed kidney-protective outcomes in patients with type 2 diabetes and chronic kidney disease[4].

TRIAL / PHASE 3 / 2024

FLOW

POPULATION
3,533 adults with T2DM and CKD (eGFR 25–75 mL/min/1.73 m², with albuminuria)
DOSE
subcutaneous 1.0 mg once weekly
DURATION
median 3.4 years (stopped early for efficacy)
PUBLICATION
NEJM · 2024
PRIMARY OUTCOME

Composite of kidney failure, sustained >=50% eGFR decline, kidney death, or CV death — HR 0.76 (95% CI 0.66–0.88, P=0.0003). CV events HR 0.82, CV death HR 0.71, all-cause mortality HR 0.80[4].

A prespecified subgroup analysis published in the European Heart Journal found that the cardiovascular benefit was consistent across all KDIGO risk strata — overall CV-event HR 0.82 (95% CI 0.68–0.98), with stratum-specific HRs of 0.67–0.84 and no significant interaction[16]. In other words, the benefit did not drop off in patients with more advanced CKD. FDA approval for the kidney/CV-death indication followed in 2025.

SELECT — cardiovascular outcomes in obesity without diabetes (2023)

SELECT (Semaglutide Effects on Cardiovascular Outcomes in People with Overweight or Obesity) was the trial that broadened the cardiovascular case for GLP-1 therapy beyond the diabetes population[3].

TRIAL / PHASE 3 / 2023

SELECT

POPULATION
17,604 adults aged >=45 with established CVD and BMI >=27, without diabetes
DOSE
subcutaneous 2.4 mg once weekly
DURATION
mean ~40 months
PUBLICATION
NEJM 2023 · 389:2221–2232
PRIMARY OUTCOME

Composite of CV death, nonfatal MI, or nonfatal stroke: 6.5% semaglutide vs 8.0% placebo — HR 0.80 (95% CI 0.72–0.90, P<0.001). All-cause mortality HR 0.81; heart failure composite HR 0.82; mean weight loss 9.4%[3].

A subsequent 208-week analysis published in Nature Medicine in 2024 documented a sustained mean weight reduction of −10.2%, with 67.8% of participants achieving >=5% weight loss and 44.2% achieving >=10% — the longest randomized weight-loss follow-up for a GLP-1 RA to date[15]. The SELECT data drove the March 2024 FDA approval for cardiovascular risk reduction in obesity without diabetes.

Abstract line-graph illustration in slate-blue, taupe, and terracotta
Schematic / overlapping hazard-ratio plots across trial programs

STEP — weight management

The STEP (Semaglutide Treatment Effect in People with Obesity) program established the chronic-weight-management indication.

TRIAL / PHASE 3 / 2021

STEP 1

POPULATION
1,961 adults with BMI >=30 (or >=27 with comorbidity), no diabetes
DOSE
subcutaneous 2.4 mg once weekly
DURATION
68 weeks
PUBLICATION
NEJM · 2021
PRIMARY OUTCOME

Mean body weight change −14.9% vs −2.4% placebo; treatment difference −12.4 percentage points (P<0.001). 86% achieved >=5% loss; 50% achieved >=15%[2].

TRIAL / PHASE 3 / 2022

STEP 5 · two-year durability

POPULATION
304 adults with overweight/obesity, no diabetes
DOSE
subcutaneous 2.4 mg once weekly
DURATION
104 weeks
PUBLICATION
Nature Medicine · 2022
PRIMARY OUTCOME

Mean weight change at week 104: −15.2% semaglutide vs −2.6% placebo (P<0.0001). 77% achieved >=5% loss vs 34% placebo[8].

TRIAL / PHASE 3 / HEAD-TO-HEAD

STEP 8 · semaglutide vs liraglutide

POPULATION
338 adults with overweight/obesity, no diabetes
DOSE
semaglutide 2.4 mg weekly vs liraglutide 3.0 mg daily
DURATION
68 weeks
PUBLICATION
JAMA · 2022 · 327(2):138–150
PRIMARY OUTCOME

Mean body weight change −15.8% semaglutide vs −6.4% liraglutide vs −1.9% placebo; treatment difference semaglutide vs liraglutide −9.4 percentage points (95% CI −12.0 to −6.8, P<0.001)[7].

TRIAL / PHASE 3 / PEDIATRIC

STEP TEENS

POPULATION
201 adolescents aged 12 to <18 with BMI >=95th percentile
DOSE
subcutaneous 2.4 mg once weekly
DURATION
68 weeks
PUBLICATION
NEJM · 2022 · 387:2245–2257
PRIMARY OUTCOME

Mean BMI change −16.1% semaglutide vs +0.6% placebo (P<0.001). 73% of semaglutide arm achieved >=5% weight loss; ~45% dropped below the BMI cutoff for clinical obesity[9].

TRIAL / PHASE 3 / HFpEF

STEP-HFpEF

POPULATION
529 adults with symptomatic HFpEF (LVEF >=45%) and BMI >=30, no diabetes
DOSE
subcutaneous 2.4 mg once weekly
DURATION
52 weeks
PUBLICATION
NEJM · 2023
PRIMARY OUTCOME

Change in KCCQ-CSS: +16.6 semaglutide vs +8.7 placebo (estimated treatment difference +7.8 points; 95% CI 4.8–10.9, P<0.001). Mean body weight change −13.3% vs −2.6%[10].

SUSTAIN and PIONEER — type 2 diabetes

The original SUSTAIN program (SUSTAIN 1–5) covered the spectrum of type 2 diabetes treatment scenarios — treatment-naive (SUSTAIN 1), background metformin or TZD (SUSTAIN 2), exenatide ER comparator (SUSTAIN 3), insulin glargine comparator (SUSTAIN 4), and basal insulin add-on (SUSTAIN 5). Across these trials, HbA1c reductions ranged from 0.7% to 2.5% on 0.5 mg and from 0.9% to 2.8% on 1.0 mg, with body weight reductions of 3.5–6.5 kg[13].

TRIAL / CV OUTCOMES / 2016

SUSTAIN-6

POPULATION
3,297 adults with T2DM at high CV risk
DOSES
subcutaneous 0.5 mg or 1.0 mg once weekly
DURATION
104 weeks
PUBLICATION
NEJM · 2016
PRIMARY & SAFETY OUTCOMES

MACE composite 6.6% semaglutide vs 8.9% placebo, HR 0.74 (95% CI 0.58–0.95, P<0.001 for noninferiority). Diabetic retinopathy complications HR 1.76 (95% CI 1.11–2.78, P=0.02) — reflected in the FDA prescribing information[1].

BOXED WARNING / RETINOPATHY

The SUSTAIN-6 signal for diabetic retinopathy complications appears to be driven by rapid HbA1c reduction in patients with pre-existing retinopathy rather than direct semaglutide toxicity. Coordinated ophthalmologic monitoring is recommended in patients with established retinopathy who start GLP-1 therapy.

TRIAL / HEAD-TO-HEAD / 2018

SUSTAIN 7 · semaglutide vs dulaglutide

POPULATION
1,201 adults with T2DM on background metformin
DOSES
semaglutide 0.5/1.0 mg vs dulaglutide 0.75/1.5 mg weekly
DURATION
40 weeks
PUBLICATION
Lancet Diabetes & Endocrinology · 2018
PRIMARY OUTCOME

Mean HbA1c reduction: 1.5% (sema 0.5 mg) vs 1.1% (dula 0.75 mg); 1.8% (sema 1.0 mg) vs 1.4% (dula 1.5 mg). Significantly greater weight loss at both doses[6].

TRIAL / ORAL / CV SAFETY / 2019

PIONEER 6

POPULATION
3,183 adults with T2DM at high CV risk
DOSE
oral 14 mg once daily
DURATION
median 15.9 months
PUBLICATION
NEJM · 2019
PRIMARY & SECONDARY OUTCOMES

MACE HR 0.79 (95% CI 0.57–1.11, P<0.001 noninferiority). All-cause mortality 1.4% vs 2.8% placebo (HR 0.51, P=0.008); CV death HR 0.49[5].

TRIAL / ORAL HIGH-DOSE / 2023

PIONEER PLUS

POPULATION
1,606 adults with T2DM on 1–3 oral agents
DOSES
oral 14 mg, 25 mg, 50 mg daily
DURATION
52 weeks
PUBLICATION
The Lancet · 2023
PRIMARY OUTCOME

HbA1c reduction −1.5% (14 mg), −1.8% (25 mg), −2.0% (50 mg). Body weight reduction −4.4 kg, −6.7 kg, −8.0 kg respectively[20].

Emerging research: alcohol use disorder

Beyond the metabolic indications, an exploratory area worth flagging is substance use. A single-site Phase 2 randomized clinical trial published in JAMA Psychiatry in 2025 tested low-dose semaglutide in 48 non-treatment-seeking adults with alcohol use disorder. The titration schedule was 0.25 mg weekly for four weeks, then 0.5 mg weekly for four weeks, then 1.0 mg for one week — a 9-week protocol[12].

TRIAL / PHASE 2 / 2025

Semaglutide in AUD

POPULATION
48 non-treatment-seeking adults with alcohol use disorder
DOSE
titration 0.25 → 0.5 → 1.0 mg weekly
DURATION
9 weeks
PUBLICATION
JAMA Psychiatry · 2025 · 82(4):395–405
SIGNALS

Significant reductions in laboratory grams of alcohol consumed, peak breath alcohol concentration, weekly alcohol craving, and heavy drinking days, with medium-to-large effect sizes. Reductions in cigarettes per day in smokers[12].

This is a small Phase 2 study, not a regulatory submission, but it has spurred larger trials of GLP-1 RAs in addiction medicine — a research arc to watch.

FDA approval timeline

Abstract horizontal timeline illustration with geometric milestone markers
Schematic / FDA approval milestones
YEARINDICATIONBASIS
Dec 2017Glycemic control, T2DM (subcutaneous, 0.5 / 1.0 mg)SUSTAIN program
Sep 2019Glycemic control, T2DM (oral, 7 / 14 mg)PIONEER program
Jun 2021Chronic weight management (adults, BMI >=30 or >=27 + comorbidity)STEP program
Dec 2022Chronic weight management (adolescents 12+)STEP TEENS
Mar 2024CV risk reduction (obesity/overweight, no diabetes)SELECT
2025Kidney/CV-death risk reduction (T2DM with CKD)FLOW
2025MASH with moderate-to-advanced fibrosis (submission, Priority Review)ESSENCE